منابع مشابه
A cause for childhood ataxia
Genetic studies uncover a mutation in a widely conserved protein as the cause of a neurological disorder in two brothers.
متن کاملAtaxia in Childhood — Symptoms and Treatment
Ataxia can be classified according to the onset into episodic, acute, intermittent and chronic ataxia. Acute ataxia in children is caused by CNS tumors, trauma, CNS infection, toxins, metabolic causes or stroke. Recurrent ataxia can be due to metabolic causes, seizures, basilar artery migraine or toxins. Chronic ataxia is usually the result of hereditary ataxia, CNS tumors, congenital anomalies...
متن کاملAtaxia in Childhood — Symptoms and Treatment
Ataxia can be classified according to the onset into episodic, acute, intermittent and chronic ataxia. Acute ataxia in children is caused by CNS tumors, trauma, CNS infection, toxins, metabolic causes or stroke. Recurrent ataxia can be due to metabolic causes, seizures, basilar artery migraine or toxins. Chronic ataxia is usually the result of hereditary ataxia, CNS tumors, congenital anomalies...
متن کاملAtaxia in Childhood — Symptoms and Treatment
Ataxia can be classified according to the onset into episodic, acute, intermittent and chronic ataxia. Acute ataxia in children is caused by CNS tumors, trauma, CNS infection, toxins, metabolic causes or stroke. Recurrent ataxia can be due to metabolic causes, seizures, basilar artery migraine or toxins. Chronic ataxia is usually the result of hereditary ataxia, CNS tumors, congenital anomalies...
متن کاملNotes on cerebellar ataxia in childhood.
The syndrome of cerebellar ataxia is one with which paediatricians are familiar not only because it is the usual presenting feature of the subtentorial tumours of childhood, but also because there is a small but well-recognized group of cases in which cerebellar ataxia is the only ultimate diagnosis. It is not proposed to catalogue all the causes of cerebellar ataxia in childhood but rather to ...
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ژورنال
عنوان ژورنال: Journal of Child Neurology
سال: 2012
ISSN: 0883-0738,1708-8283
DOI: 10.1177/0883073812448840